Chemical burns result from skin or eye contact with corrosive substances causing progressive tissue destruction until neutralized. Acids cause coagulation necrosis creating firm eschar limiting penetration depth. Alkalis cause liquefaction necrosis penetrating deeper with ongoing damage. Common causative agents include industrial chemicals, household cleaners, battery acid, and cement. Severity depends on chemical strength, concentration, duration, and body area affected. Immediate treatment requires copious water irrigation for 20-30 minutes minimum. Specific antidotes exist for some chemicals like hydrofluoric acid requiring calcium gluconate. Delayed presentation occurs with some chemicals appearing minor initially then worsening. Eye exposure threatens vision requiring immediate irrigation and ophthalmology consultation. Inhalation creates airway burns requiring intubation and specialized management. Systemic toxicity from absorption complicates some chemical burns beyond local damage. Workplace exposures implicate employer safety failures and OSHA violations. Product liability applies when inadequate warnings or defective containers cause exposure. Intentional attacks using chemicals constitute assault with enhanced criminal penalties. Long-term effects include scarring, contractures, and increased cancer risks. Understanding chemical burn uniqueness helps pursue appropriate treatment and compensation for these preventable injuries.